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Gustaf Retzius and Asjep Gley compounded his research, the latter credited with the discovery of the function of the parathyroid glands.

Osteitis fibrosa cystica is defined as the classic skeletal manifestation of advanced hyperparathyroidism. The addition of weight loss, appetite lossvomiting, polyuriaand polydipsia to the aforementioned symptoms may indicate that OFC is the result of parathyroid carcinoma.

Abnormalities affecting the parathyroid glands cause a surplus of PTH, which, in turn, increases the activity and frequency of osteoblasts and osteocytes. Muscles in patients afflicted with OFC can either appear unaffected or “bulked up. The first published literature to describe a brown tumor which was linked to OFC was published inthough clinical reports from before do draw a correlation between the disease and tumors previous to the publication. Almost all who undergo parathyroidectomy experience increased bone density and repair of the skeleton within weeks.

Fuller Albright diagnosed and treated the woman, who suffered from a large tumor of the neck as well as renal stones. Paget’s disease of bone. Clinical Orthopaedics and Related Research. Primary Secondary Tertiary Osteitis fibrosa cystica. Osteitis fibrosa cystica is the result of unchecked hyperparathyroidism, or the overactivity of the parathyroid glandswhich results in an overproduction of parathyroid hormone PTH. The discovery and subsequent description of the parathyroid glands is credited to Ivar Sandstrom, though his publication, On a New Gland in Man and Several Mammals-Glandulae Parathyroideaereceived little attention.


National Library of Medicine.

Blood tests on patients with OFC generally show high levels of calcium normal levels are considered to range between 8. In especially severe cases of OFC, parathyroidectomy, or the full removal of the parathyroid glands, is the chosen route of treatment.

Endocrine, nutritional, and metabolic diseases”.

Medical management of OFC consists of Vitamin D treatment, generally alfacalcidol or calcitrioldelivered intravenously. From Wikipedia, the free encyclopedia. Fine needle aspiration FNA can be used to biopsy bone lesionsonce found on an X-ray or other scan. Osteitis fibrosa cystica Asskep fibrosa cystica of the tibia. Symptoms are boiter often more severe. Cysts may be lined by osteoclasts and sometimes blood pigments, which lend to the notion of “brown tumors.

Nucleoli also tend to be smaller than average. X-rays may also be used to diagnose the disease. Osteitis fibrosa cystica has qskep been a rare disease. Journal of Medical Case Reports. The effects of OFC on bone are largely dependent on the duration of the disease and the level of parathyroid hormone PTH produced.

Hypofunction Diabetes mellitus types: Diseases of the endocrine system E00—E35— The hospitalization rate for hyperparathyroidism in the United States in was 8.


Osteitis fibrosa cystica

The hyperparathyroidism can be triggered by a parathyroid adenomahereditary factors, parathyroid carcinomaor renal osteodystrophy. Bone and joint disease M80—M94— Osteoclastic bone resorption releases minerals, including calciumfrom the bone into the bloodstream, goirer both elevated blood calcium levels, and the structural changes which weaken the bone. Archived PDF from the original on Cushing’s syndrome Pseudo-Cushing’s syndrome sex hormones: Disorders such as familial hyperparathyroidism, multiple endocrine neoplasia type 1 MEN Type 1 and hyperparathyroidism-jaw tumor syndrome can, if left unchecked, result in OFC.

Such tests can be vital in diagnosis and can also prevent unnecessary treatment and invasive surgery. The post-surgical survival rate hovers around seven years, while patients who do not undergo surgery have a survival rate of around five goiher. When calcitriol levels decrease, parathyroid hormone levels increase, halting the storage of calcium, and instead triggering its removal from the bones.

Osteitis fibrosa cystica – Wikipedia

The Western Journal of Medicine. Approximately 1 in 10 documented cases of hyperparathyroidism are a result of hereditary factors. The condition was first described by Gerhard Engel in and Friedrich Daniel von Recklinghausen inthough William Hunterwho died inis credited with finding the first example of gkiter disease.